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COVID-19 Pandemic: Transmissions, Deaths, Treatments, Vaccines, Interventions and More...

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Only if you're not within 6 feet of others. The sun's UV rays will not instantly deactivate virus particles coming from another person's sneeze/cough/breath in the second or two it takes for them to reach you, assuming they're close by.
Of all the things that has been solid about this virus from day 1 is exactly that. The sun's rays seem to squash it within seconds. Now we just need some sun.
 
Cherry picked? They picked all the data, essentially. Everyone who was given HCQ/CQ within 48 hours of admission (to exclude more severely ill patients who had been in hospital longer, as well as excluding anyone whose HCQ/CQ treatment began while on a ventilator) vs. almost everybody else who was admitted. Hard to be biased when you're not excluding a bunch of patients (and they did exclude remdesivir from the "control" group to make sure that didn't bias that group). This is 15K patients treated vs. 80K patients not treated - this ain't a 20 person anecdotal retrospective study.

And if you think it's cherry picked the burden of proof is on you. Show your work. Tell us what you've figured out that nobody else has. Pretty sure you can't. It's over. HCQ is not only ineffective, it's dangerous in a hospital setting with higher mortality rates. It doesn't get much worse than the summary below from the authors. Maybe there will be some prophylactic miracle, but I highly doubt that either, given its track record, so far.

In summary, this multinational, observational, real-world study of patients with COVID-19 requiring hospitalisation found that the use of a regimen containing hydroxychloroquine or chloroquine (with or without a macrolide) was associated with no evidence of benefit, but instead was associated with an increase in the risk of ventricular arrhythmias and a greater hazard for in-hospital death with COVID-19. These findings suggest that these drug regimens should not be used outside of clinical trials and urgent confirmation from randomised clinical trials is needed.
Were these studies randomized? Was zinc included? As I said I’ll wait for a double blinded study of HCQ with Azithromycin and zinc, given in the early stages of infection. Happy to admit HCQ has no benefit once that study is conducted and shows no benefit. I hope you’ll be able to admit you’re wrong, if and when you are.
 
Just not understanding how beaches are open and people allowed to lay around/walk near each other for hours on end with no mask "mandate" or requirements.
1)Pretty sure it's a thing that outdoor transmission of the virus has been shown to be significantly less. Especially in the sun, especially if over 76 degrees(read that was a magic # of sorts). Sunny and 76+ is pretty much prerequisite for large crowds at the beach. 76 degrees is actually very low for a packed beach day.

2)I think it is much easier to maintain a proper social distance at the beach as opposed to a store. Many of the pictures I've seen of people at the beach which have drawn criticism are a) deceiving because they are shot with a long lens which condenses the figures in the picture, b)not that crazy.
 
Reading all this stuff on HCQ I'm coming to the conclusion that it helps a little.

So not worthy of the hype from the pres, and not worthy of the condemnation from the left.
 
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Same here, so we should both be fine - however your post didn't specify any of what you just said...
Grew up in Lavallette. In my entire life I sat on the beach for a sun tan is less than 10 days. F-ing boring. Now fishing, surfing, body surfing, and sailing a Hobie Cat is a completely different story. Mostly just fish and surf now so social distancing is not an issue.
 
Of all the things that has been solid about this virus from day 1 is exactly that. The sun's rays seem to squash it within seconds. Now we just need some sun.

+1
Sun kills the virus within seconds. Go to the beach on a sunny day. :)

Wrong and wrong. Takes almost 7 minutes to deactivate 90% of SARS-CoV-2 with simulated summer soltice 40N sunlight (best case), meaning almost none of it would be deactivated in the 1-2 seconds it takes virus-laden droplets to go from person A to person B. Does anybody here actually look anything up? I could spend all day just correcting misinformation...

https://academic.oup.com/jid/advance-article/doi/10.1093/infdis/jiaa274/5841129

Previous studies have demonstrated that SARS-CoV-2 is stable on surfaces for extended periods under indoor conditions. In the present study, simulated sunlight rapidly inactivated SARS-CoV-2 suspended in either simulated saliva or culture media and dried on stainless steel coupons. Ninety percent of infectious virus was inactivated every 6.8 minutes in simulated saliva and every 14.3 minutes in culture media when exposed to simulated sunlight representative of the summer solstice at 40oN latitude at sea level on a clear day.
 
Wrong and wrong. Takes almost 7 minutes to deactivate 90% of SARS-CoV-2 with simulated summer soltice 40N sunlight (best case), meaning almost none of it would be deactivated in the 1-2 seconds it takes virus-laden droplets to go from person A to person B. Does anybody here actually look anything up? I could spend all day just correcting misinformation...

https://academic.oup.com/jid/advance-article/doi/10.1093/infdis/jiaa274/5841129

Previous studies have demonstrated that SARS-CoV-2 is stable on surfaces for extended periods under indoor conditions. In the present study, simulated sunlight rapidly inactivated SARS-CoV-2 suspended in either simulated saliva or culture media and dried on stainless steel coupons. Ninety percent of infectious virus was inactivated every 6.8 minutes in simulated saliva and every 14.3 minutes in culture media when exposed to simulated sunlight representative of the summer solstice at 40oN latitude at sea level on a clear day.

I vote we haul him off to the vet and have him put out of our misery.
 
Wrong and wrong. Takes almost 7 minutes to deactivate 90% of SARS-CoV-2 with simulated summer soltice 40N sunlight (best case), meaning almost none of it would be deactivated in the 1-2 seconds it takes virus-laden droplets to go from person A to person B. Does anybody here actually look anything up? I could spend all day just correcting misinformation...

https://academic.oup.com/jid/advance-article/doi/10.1093/infdis/jiaa274/5841129

Previous studies have demonstrated that SARS-CoV-2 is stable on surfaces for extended periods under indoor conditions. In the present study, simulated sunlight rapidly inactivated SARS-CoV-2 suspended in either simulated saliva or culture media and dried on stainless steel coupons. Ninety percent of infectious virus was inactivated every 6.8 minutes in simulated saliva and every 14.3 minutes in culture media when exposed to simulated sunlight representative of the summer solstice at 40oN latitude at sea level on a clear day.
You are talking about saliva. I'm talking about normal breathing. Big difference.
 
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Were these studies randomized? Was zinc included? As I said I’ll wait for a double blinded study of HCQ with Azithromycin and zinc, given in the early stages of infection. Happy to admit HCQ has no benefit once that study is conducted and shows no benefit. I hope you’ll be able to admit you’re wrong, if and when you are.
We're not talking about early stages of infection, as you just said you think the Lancet study was essentially forged and the Lancet study was post hospital admission (within 48 hours, which are generally not "severe" cases but moderate ones). You can't "randomize" a retrospective study, per se, but a 90,000 patient study is probably impossible to fake (and this wasn't some guy in his garage - it was done by some of the most respected people in medical science) and the evidence is simply overwhelming. There would have to be monumental bias to even to move the needle from "HCQ kills 30-45% more patients than non-HCQ" to "HCQ is about the same as non-HCQ."

I'm still open-minded, but very skeptical of HCQ used as a prophylactic/post-exposure prophylactic or very mildly ill (not hospitalized) patients, mostly because it's going to take huge numbers of patients to detect what is likely to be a tiny difference, given that the vast majority (80-85%) of infected people get better on their own. I'm already not wrong on HCQ for hospitalized and worse patients - that's blindingly obvious now. And I highly doubt I'll be wrong on mildly ill patients. But if I am, I'll own up to it.
 
You are talking about saliva. I'm talking about normal breathing. Big difference.
Sunlight is not going to be more than marginally faster at deactivating viruses in breath vs. larger droplets, plus droplets are the biggest fear, since they come from coughs/sneezes, which we know have far higher viral loads in them vs. breath. People likely are reading about how fast UVC radiation is at deactivating viruses (and it's much faster than simulated surface sunlight) and misapplying that to sunlight, which has all of its UVC radiation absorbed by the ozone layer, such that none of it reaches the earth's surface.
 
Grew up in Lavallette. In my entire life I sat on the beach for a sun tan is less than 10 days. F-ing boring. Now fishing, surfing, body surfing, and sailing a Hobie Cat is a completely different story. Mostly just fish and surf now so social distancing is not an issue.
Same here - I hate sitting on the beach, partly because I burn easily and hate putting on sunscreen, so I mostly go before 10 am or after 4 pm. When I do go, it's mostly to body surf, which my son and I really enjoy. I'll gladly put sunscreen on to play golf, though, lol...
 
Sunlight is not going to be more than marginally faster at deactivating viruses in breath vs. larger droplets, plus droplets are the biggest fear, since they come from coughs/sneezes, which we know have far higher viral loads in them vs. breath. People likely are reading about how fast UVC radiation is at deactivating viruses (and it's much faster than simulated surface sunlight) and misapplying that to sunlight, which has all of its UVC radiation absorbed by the ozone layer, such that none of it reaches the earth's surface.
Aren't droplets much larger than airborne particles by a huge percentage?? I mean by a lot.
 
Yeah, I'll take the Indian government's word without any paper associated with it over the Lancet's analysis of ~15K HCQ/CQ patients (vs. 80K non-HCQ/CQ patients), who all started treatment within 48 hours of admission to the hospital. Stop looking for needles in haystacks and pay attention to the big picture here.
Frankly I am tired of your schtick. I am not reaching, I read the study and see they failed to include data to assess disease severity. You read DL's blog.

If you can't see the flaws in that paper then you are blind. Patients spanning back to last December when they did not even know what they were treating or how to treat. Up until 6 weeks ago we were killing patients with an incorrect ventilation approach. 48 hrs after hospitalization is roughly 15 days of disease course which is no different then almost every other retrospective study that shows little benefit. Also, hospitalization by definition means the course was severe. Mild are those who are either symptomless or nearly so. Moderate is I can tough it at home. Hospitalization is the 3% who can't breath and are in serious trouble.

They used qSOFA to determine severity LOLOLOLOL.
sfIBczVFH24BjZ2uoptxYhz6CDf0T2k-8zaJiHfeRPiMjxmnp1IL9zwKB9HziYw1R3JyqargXdzGgf886FWdBMbkWUwFplzTadGDqpPMAKsGXibrYBxCVTL3e8NoFXJTqhfZ6QGp

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7167215/
We anticipate that the qSOFA is not appropriate to identify Covid-19 patient to have poor outcomes typical of sepsis.

Also, where is the min, max, mean of SPO? If you have two groups of 1000 and both have 50% with patients below 94%. Are they equivalent populations if one has min, max, mean of 50, 96 and 71 and the other 88, 98, 91?

That is how you manipulate data to say what you want it too. Please remember what I have been doing for 20 years. I see it all the time.

However, since all you can do is read DL blog, I will help you understand the data a little better with a free insight:

LWenpyhNqTnXySXmlbH22X00GcXLnrPNdPmR8cKV6qgj-lzYj9at_qEIP0U9jQL4XctQbfyCrp-rUgwgpwNvW6nVgzr2jl5bu4fM3GNOZg8U2H3EPkjXekUYNN6RJDtbIqFCttuZ

The ICU stay was almost twice as long for treated patients vs the control and that was extremely significant. The treated patients needed mechanical ventilation at a rate of 3X the amount in the control. Again an EXTREMELY significant difference between treated and control. Conclusion, the treated patients were worse off. Which is what has happened in every retrospective study so far. Logically, it makes sense. If someone's IL-6 and CRP indicate extreme situation naturally doctors start trying hail mary's. I would bet you any sum of money the IL-6, CRP, etc of the treated group was SIGNIFICANTLY worse than the control.

The Indian Govt did a study on several thousand workers and made decisions that impact many thousands of lives directly based on the results. Just because they have not shared that data does not mean anything other than they are not sharing it. The Lancet paper is retrospective with no data on disease severity and itself admits its limitations "Nevertheless, a cause-and-effect relationship between drug therapy and survival should not be inferred."
 
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Wrong and wrong. Takes almost 7 minutes to deactivate 90% of SARS-CoV-2 with simulated summer soltice 40N sunlight (best case), meaning almost none of it would be deactivated in the 1-2 seconds it takes virus-laden droplets to go from person A to person B. Does anybody here actually look anything up? I could spend all day just correcting misinformation...

https://academic.oup.com/jid/advance-article/doi/10.1093/infdis/jiaa274/5841129

Previous studies have demonstrated that SARS-CoV-2 is stable on surfaces for extended periods under indoor conditions. In the present study, simulated sunlight rapidly inactivated SARS-CoV-2 suspended in either simulated saliva or culture media and dried on stainless steel coupons. Ninety percent of infectious virus was inactivated every 6.8 minutes in simulated saliva and every 14.3 minutes in culture media when exposed to simulated sunlight representative of the summer solstice at 40oN latitude at sea level on a clear day.


is someone coughing up a lung in large concentration, how would you catch it if just a tiny tiny amount, you want to argue the a small point rather than the bottom line
 
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Don't want a hero's badge at all.
Dont mind putting on a mask to benefit others and doing my part (which is very easy and takes minimal effort).
I'm certainly not doing anything extraordinary.

Just not understanding how beaches are open and people allowed to lay around/walk near each other for hours on end with no mask "mandate" or requirements.


because outside is the safest place to be, pretty much common sense, you dont need a mask walking in the outdoors, but continue to do you
 
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Same here - I hate sitting on the beach, partly because I burn easily and hate putting on sunscreen, so I mostly go before 10 am or after 4 pm. When I do go, it's mostly to body surf, which my son and I really enjoy. I'll gladly put sunscreen on to play golf, though, lol...
It's funny and at the time same lucky. I rarely used sunscreen most of my life. Of Irish descent and once you get that first burn and peeling than you tan. Once it gets nice out I spend all my time in the sun constantly. Especially my days playing golf and on the range from sun up to sun down. Or bike riding for hours on end. Today I lotion up a lot except fishing. Fish don't bite if you are wearing Coppertone. They can smell that. No issues so far
 
Reading all this stuff on HCQ I'm coming to the conclusion that it helps a little.

So not worthy of the hype from the pres, and not worthy of the condemnation from the left.
It's not the left, it's science. C'mon. But agree it was never worth any hype - nothing really is without controlled trials, outside of certain observational trials that have overwhelmingly positive or negative results - this can sometimes be seen in very binary trials like antbiotics where often either it works or doesn't and "works" is fairly easy to measure.
 
It's not the left, it's science. C'mon. But agree it was never worth any hype - nothing really is without controlled trials, outside of certain observational trials that have overwhelmingly positive or negative results - this can sometimes be seen in very binary trials like antbiotics where often either it works or doesn't and "works" is fairly easy to measure.
No I meant the condemnation from the left.

The science seems to say "meh".
 
It's not the left, it's science. C'mon. But agree it was never worth any hype - nothing really is without controlled trials, outside of certain observational trials that have overwhelmingly positive or negative results - this can sometimes be seen in very binary trials like antbiotics where often either it works or doesn't and "works" is fairly easy to measure.
It is not science. qSOFA as the severity indicator. LOL.
 
Frankly I am tired of your schtick. I am not reaching, I read the study and see they failed to include data to assess disease severity. You read DL's blog.

If you can't see the flaws in that paper then you are blind. Patients spanning back to last December when they did not even know what they were treating or how to treat. Up until 6 weeks ago we were killing patients with an incorrect ventilation approach. 48 hrs after hospitalization is roughly 15 days of disease course which is no different then almost every other retrospective study that shows little benefit. Also, hospitalization by definition means the course was severe. Mild are those who are either symptomless or nearly so. Moderate is I can tough it at home. Hospitalization is the 3% who can't breath and are in serious trouble.

They used qSOFA to determine severity LOLOLOLOL.
sfIBczVFH24BjZ2uoptxYhz6CDf0T2k-8zaJiHfeRPiMjxmnp1IL9zwKB9HziYw1R3JyqargXdzGgf886FWdBMbkWUwFplzTadGDqpPMAKsGXibrYBxCVTL3e8NoFXJTqhfZ6QGp

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7167215/
We anticipate that the qSOFA is not appropriate to identify Covid-19 patient to have poor outcomes typical of sepsis. Also, where is the min, max, mean of SPO? If you have two groups of 1000 and both have 50% with patients below 94%. Are they equivalent populations if one has min, max, mean of 50, 96 and 71 and the other 88, 98, 91?

That is how you manipulate data to say what you want it too. Please remember what I have been doing for 20 years. I see it all the time.

However, since all you can do is read DL blog, I will help you understand the data a little better with a free insight:

LWenpyhNqTnXySXmlbH22X00GcXLnrPNdPmR8cKV6qgj-lzYj9at_qEIP0U9jQL4XctQbfyCrp-rUgwgpwNvW6nVgzr2jl5bu4fM3GNOZg8U2H3EPkjXekUYNN6RJDtbIqFCttuZ

The ICU stay was twice as long for treated patients vs the control and that was extremely significant. The treated patients needed mechanical ventilation at a rate of 3X the amount in the control. Again an EXTREMELY significant difference between treated and control. Conclusion, the treated patients were worse off. Which is what has happened in every retrospective study so far. Logically, it makes sense. If someone's IL-6 and CRP indicate extreme situation naturally doctors start trying hail mary's. I would bet you any sum of money the IL-6, CRP, etc of the treated group was SIGNIFICANTLY worse than the control.

The Indian Govt did a study on several thousand workers and made decisions that impact many thousands of lives directly based on the results. Just because they have not shared that data does not mean anything other than they are not sharing it. The Lancet paper is retrospective with no data on disease severity and itself admits its limitations "Nevertheless, a cause-and-effect relationship between drug therapy and survival should not be inferred."
+1
You hit the nail on the head.....once again. Thanks for posting the real story.
 
Copper fit masks?

Copper does have virus killing properties right?

Does copper kill it fast enough, and is there enough copper in it to make a difference?

Both funny and interesting imo.
 
Aren't droplets much larger than airborne particles by a huge percentage?? I mean by a lot.
The virus doesn't change size depending on how it's expelled. A virus particle is a virus particle. Large droplets may contain considerably more virus particles, but not bigger particles. If it takes 7 mins to deactivate 1 particle, it takes 7 min. Spit or breath, doesn't matter.
 
The virus doesn't change size depending on how it's expelled. A virus particle is a virus particle. Large droplets may contain considerably more virus particles, but not bigger particles. If it takes 7 mins to deactivate 1 particle, it takes 7 min. Spit or breath, doesn't matter.
Yeah no. That is the dumbest post on this 200+ page thread.
 
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Frankly I am tired of your schtick. I am not reaching, I read the study and see they failed to include data to assess disease severity. You read DL's blog.

If you can't see the flaws in that paper then you are blind. 48 hrs after hospitalization is roughly 15 days of disease course which is no different then almost every other retrospective study that shows little benefit. Also, hospitalization by definition means the course was severe. Mild are those who are either symptomless or nearly so. Moderate is I can tough it at home. Hospitalization is the 3% who can't breath and are in serious trouble.

They used qSOFA to determine severity LOLOLOLOL.
sfIBczVFH24BjZ2uoptxYhz6CDf0T2k-8zaJiHfeRPiMjxmnp1IL9zwKB9HziYw1R3JyqargXdzGgf886FWdBMbkWUwFplzTadGDqpPMAKsGXibrYBxCVTL3e8NoFXJTqhfZ6QGp

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7167215/
We anticipate that the qSOFA is not appropriate to identify Covid-19 patient to have poor outcomes typical of sepsis.

However, since all you can do is read DL blog, I will help you understand the data a little better with a free insight:

LWenpyhNqTnXySXmlbH22X00GcXLnrPNdPmR8cKV6qgj-lzYj9at_qEIP0U9jQL4XctQbfyCrp-rUgwgpwNvW6nVgzr2jl5bu4fM3GNOZg8U2H3EPkjXekUYNN6RJDtbIqFCttuZ

The ICU stay was twice as long for treated patients vs the control and that was extremely significant. The treated patients needed mechanical ventilation at a rate of 3X the amount in the control. Again an EXTREMELY significant difference between treated and control. Conclusion, the treated patients were worse off. Which is what has happened in every retrospective study so far. Logically, it makes sense. If someone's IL-6 and CRP indicate extreme situation naturally doctors start trying hail mary's. I would bet you any sum of money the IL-6, CRP, etc of the treated group was SIGNIFICANTLY worse than the control.

The Indian Govt did a study on several thousand workers and made decisions that impact many thousands of lives directly based on the results. Just because they have not shared that data does not mean anything other than they are not sharing it. The Lancet paper is retrospective with no data on disease severity and itself admits its limitations "Nevertheless, a cause-and-effect relationship between drug therapy and survival should not be inferred."

Same here, very tired of your schtick. I have 95% of medical science on my side and you have the discredited Didier Raoult, Elon Musk and Dr. Trump on your side. HCQ is simply not effective and is actually dangerous for hospitalized COVID patients. You simply can't see the forest for the trees and keep looking for minor differences that don't really mean anything when all you have to look at is that the HCQ and comparators were matched pretty damn closely and the mortality rate was significantly greater for the HCQ patients. For a huge sample size of 15K/80K.

Don't you think that if HCQ had any efficacy at all there would have at least been some signal in a huge dataset like that? And maybe the ICU stays were longer and ventilation needs were greater for the HCQ patients because HCQ was actually worse for patients than the control - hence the eventual greater mortality rate as an outcome.

And yes, I misspoke on moderate - should have simply said less severe vs. more severe, as they did exclude the most severe patients (intubated) at start of HCQ treatment from the HCQ data, but you're correct that it's generally at least severe to be admitted to the hospital. By the way about 20% of COVID cases end up in the hospital, not 3%. And I didn't even bring Lowe's blog up today, although since you asked, he savaged HCQ in it today and rightly so.

https://blogs.sciencemag.org/pipeline/archives/2020/05/22/hydroxychloroquine-enough-already

From Lowe's Blog: But I’m going to leave it at this. There was no evidence whatsoever of any benefit with any of these treatment regimes. There was significant evidence of harm. Here’s how it works: when something is real, you continue to see a real signal as you collect more and better data. When something is not real, it disappears. Tell me again why anyone should be advocating such treatments. But your reasons had better stand up to 14,888 patients versus 81,144 comparators. Make it good.

At this point, there's probably not much more productive from continuing this conversation. Let's see what happens with the prophylactic and mild symptom studies, although I'm surprised you're simply going to buy the reports from India on this without a scientific paper/description behind it. The only published data I've seen so far on prevention of getting COVID has been from lupus patients on HCQ, who are significantly more likely to get COVID than RA patients. Granted they're not "typical" patients, but that's certainly not a good sign for prevention.
 
I would say 75% of people I see in public are wearing masks (outside establishments).

You might be too “tough” to wear a mask, but stop making $hit up.

You’re a terrible troll.
75% is a fantasy. Be honest and get real. Let's keep this thread about the facts. 10% at most. Look at recent photos from NJ beaches and parks.
 
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